{
    "uniprot_canonical_ac": "A5PLK6-1", 
    "publication_count": 6, 
    "reported_interactions": 0, 
    "record_type": "protein", 
    "predicted_glycosites": 0, 
    "protein_names_refseq": "regulator of G-protein signaling like 2; regulator of G-protein signaling protein-like; regulator of G-protein signaling protein-like isoform 1", 
    "refseq_ac": "NP_001131141.1", 
    "reported_n_glycosites": 0, 
    "refseq_name": "regulator of G-protein signaling protein-like isoform 1", 
    "record_id": "A5PLK6-1", 
    "protein_names_uniprotkb": "Regulator of G-protein signaling protein-like; Regulator of G-protein-signaling protein-like", 
    "pathway": " (hsa:353299); G alpha (q) signalling events (R-HSA-416476); G alpha (i) signalling events (R-HSA-418594); G alpha (z) signalling events (R-HSA-418597)", 
    "function": "A detailed mapping of the breakpoints revealed intragenic microdeletions affecting the coding regions of RGSL2, RGSL1., [RefSeq]; Data indicate that multiplex ligation-dependent probe amplification (MLPA) probes (Fig. 2) corresponding to the RGSL2, RGSL1 and RGS16 genes showed 64.5 % tumour samples with copy number gains and 5 % tumour samples with copy number losses., [RefSeq]; Missense mutations in RGSL1 gene is associated with breast cancer., [RefSeq]; Observational study of gene-disease association. (HuGE Navigator) Publication Status: Online-Only, [RefSeq]", 
    "protein_name": "Regulator of G-protein signaling protein-like", 
    "reported_mutagensis": 0, 
    "reported_phosphosites": 0, 
    "reported_o_glycosites_with_glycan": 0, 
    "tax_id": 9606, 
    "organism": "Homo sapiens", 
    "disease": "", 
    "reported_glycation": 0, 
    "reported_snv": 12, 
    "mass": 125688.0, 
    "gene_names_uniprotkb": "RGSL1; RGSL2; RGSL", 
    "reported_n_glycosites_with_glycan": 0, 
    "reported_o_glycosites": 0, 
    "gene_names_refseq": "RGSL1; RGSL; RGSL2", 
    "gene_name": "RGSL1"
}