{
    "uniprot_canonical_ac": "A2ACT7-1", 
    "publication_count": 5, 
    "reported_interactions": 0, 
    "record_type": "protein", 
    "predicted_glycosites": 0, 
    "protein_names_refseq": "collagen alpha-3(IX) chain isoform 2 precursor; procollagen, type IX, alpha 3", 
    "refseq_ac": "NP_034066.2", 
    "reported_n_glycosites": 0, 
    "refseq_name": "collagen alpha-3(IX) chain isoform 2 precursor", 
    "record_id": "A2ACT7-1", 
    "protein_names_uniprotkb": "Collagen, type IX, alpha 3", 
    "pathway": "Collagen biosynthesis and modifying enzymes (R-MMU-1650814); Signaling by PDGF (R-MMU-186797); Assembly of collagen fibrils and other multimeric structures (R-MMU-2022090); Integrin cell surface interactions (R-MMU-216083); ECM proteoglycans (R-MMU-3000178); NCAM1 interactions (R-MMU-419037); Collagen chain trimerization (R-MMU-8948216)", 
    "function": "Results show that absence of Col IX induces early developmental, structural and biomechanical alterations in both vertebral body and intervertebral disc which eventually cause severe degenerative changes in the aging spine., [RefSeq]", 
    "protein_name": "Collagen, type IX, alpha 3", 
    "reported_mutagensis": 0, 
    "reported_phosphosites": 0, 
    "reported_o_glycosites_with_glycan": 0, 
    "tax_id": 10090, 
    "organism": "Mus musculus", 
    "disease": "multiple epiphyseal dysplasia 3 (Disease Ontology:DOID:0070304); osteochondrodysplasia (Disease Ontology:DOID:2256)", 
    "reported_glycation": 0, 
    "reported_snv": 0, 
    "mass": 63475.0, 
    "gene_names_uniprotkb": "Col9a3", 
    "reported_n_glycosites_with_glycan": 0, 
    "reported_o_glycosites": 0, 
    "gene_names_refseq": "AV006866; Col9a3", 
    "gene_name": "Col9a3"
}